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The Truth Will OUT!

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    The Truth Will OUT!

    My good news is in relation to the science around BSE - go figure!!

    First, found this study on how the use of "sonciation" allowed for a better analysis of lead in the brain.
    see:

    "Epub 2011 Sep 17.
    Ultrasonic energy as a tool to overcome some drawbacks in the determination of lead in brain tissue and urine of rats.

    Guimarães D, Santos JP, Carvalho ML, Vale G, Santos HM, Geraldes V, Rocha I, Capelo JL."

    (From abstract - "Utrasonication provided by an ultrasonic probe succeeded in extracting lead from brain samples.") it extracted more than when not doing this process, thus unveiling the lead trapped inside proteins.

    Link to pubmed/abstract:
    http://www.ncbi.nlm.nih.gov/pubmed?term=lead%20brain%20urine%20sonication

    THIS elucidates a hypothesis that when BSE contaminated brain tissue is homogenated and sonicated before injection, in transmission studies - the process of sonication is likely releasing the metals bound to the proteins and breaking apart the fibrils so their energy (from the metals) is capable of reseeding more fibrils....


    EVEN MORE IMPORTANT THOUGH:

    Dr. Vitaly Vodyanoy of Auburn University has been granted another patent. The patent gives hope in further research. The patent is:

    METHODS FOR ISOLATING PROTEONS FROM PLASMA SAMPLES
    USA Patent #8,298,793 granted October 30, 2012.

    "Abstract
    Compositions and methods for the isolation and manipulation of misfolded, or partially misfolded, proteins present in blood and other biological materials are provided. In one aspect of the invention, the compositions, hereinafter termed "proteons" are misfolded or partially misfolded proteins surrounding a metallic nanocluster, termed "proteon nucleation center" (PNC). Also provided are compositions and methods for the isolation and manipulation of proteon nucleation centers (PNCs) upon which the proteons of the present in blood and other biological materials form."

    Go to http://patft.uspto.gov/netahtml/PTO/search-bool.html
    and search the patent number, and or Dr. Vitaly Vodyanoy's name to bring up the patent.

    This document is a long read but if you go to the end pages you will find information on where all this is leading. For example:

    "Hypothesis

    Metal clusters (Gonzalez-Morega, 1993; Aiken & Finke, 1999) play a role in the seeds in proteons, Metals involved in the natural folding and misfolding of proteins are not in the ion state, but rather in the clusters of metal atoms. These atoms are directly bonded to each other creating a polyatomic metallic nucleus that can exist alone or are associated with a given number of ligands.

    Rationale:

    The most remarkable property of metal clusters is that they provide a binding template for folding and aggregation of proteins (Liu and Xu, 2002). High symmetry is one of the main characteristics of metal clusters (Gonzalez-Moraga, 1993). Packing of secondary structures prefers geometrical patterns (Gracy et al., 1993; Onuchic et al., 1997). Many disorders arise from misfolding and aggregation of an underlying protein (Carrel and Lormas, 1997; Sato, 2001). The same disorders were shown to depend on metal misbalance (Bush, 2000). Copper, iron or manganese are involved in the aging and neurological disorders. Metals are involved in prion diseases (Lehman, 2002, Thackray, et al., 2002). Among 327 possible protein folds (Chothia, et al., 1997) only 230 folds are unique. The number of unique space groups applied to inorganic clusters is also equal to 230! (Hahn, 1987). The mathematical accuracy and stability of geometrical patterns and a large number of structural arrangements of metal clusters make them uniquely qualified as perfect templates for protein folding.".....

    "What Prions are Proteons?

    Proteons and prions share many physical and chemical properties. They both are misfolded proteins, polymorphic, have no nucleic acids, metal dependent, and very resistant. They both may be induced by seeds and be controlled by guanidine hydrochloride. They both play a certain role in a cell death. They both have the same proliferation rate at 37.degree. C. (Aguzzi and Heppner, 2002; Everbroeck, et al., 2002; Ness, et al., 2002; Bounias and PURDEY, 2002; Gemer 1997, 2002). Our experiments with the recombinant human protein described in the section "Proteons from prion protein (PrP)" suggest that metal nanoclusters (PNCs) play important role in production of prion particles. We need to present experimental evidence that metal particles are indeed inside the prions particles. In order to make a statement that naturally occurring prions are proteons, that is to say, natural prion particle is the misfolded prion protein aggregated around non-organic nanoparticle that serves as a nucleating center, it would require to analyze prions obtained from the animal with the prion disease. There is substantial evidence that PrP is a Cu-binding protein. It is suggested that in conversion to the abnormal form the Cu-binding activity is switched to the binding other metals such as Mn or Zn (Brown, 2004). In this work we do not plan to work with prions obtained from animals with prion diseases. We will work with in vitro system of prion particles produced from the recombinant human PrP(23-230) protein and metal PNC and examine if these particles have acquired the ability to convert the normal form of the protein into this same abnormal (prion) form."


    There is always an ultimate goal when doing research, and it usually means finding a way to make money from what your doing. Obviously, an accurate test is being sought. But in the search for the test, comes the truth of the disease. Sadly, Dr. Vodyanoy will NOT be working with the actual diseased TSE brain tissue/samples. Why not, I ask?

    Please pass on this information to anyone that you know who will shine the light on these developments, especially those at universities, institutes, and even government employees who are doing their own investigations. The more people that know about this, the better. HAPPY NEW YEAR.

    #2
    Wasn't this stuff the same thing Mark
    Purdy was talking about some years ago?
    What you posted here is in essence saying
    that heavy metals in the brain is aiding
    in bringing about prion diseases ie. BSE?

    Comment


      #3
      Pretty simple. You don't make a hypothesis and work with the end product. You take the original product and manipulate it in order to test your hypothesis. Basic scientific method.

      Look at cancer research. You don't study cancer cells themselves because they don't follow a sequence in their development and generation. You take healthy cells, and subject them to outside influences to see what initiates the development of a healthy cell into a cancer cell. Now cancer research is so advanced that it is recognized that there are some similar factors in cancer replication, but that is not the driving focus, ie we want to prevent the cancer, not just stop it from multiplying.

      The study is looking at the influence of metals on prion development. Nothing to be learned by looking at the prions. To prove Cu is a factor, you can't just look at the end product and say, yep, that's the culprit. You better prove it by taking healthy cells and using the influence of copper concentrations, produced the end product (prion).

      So Kathy, you better start taking some classes in basic scientific inquiry before you start ranting and raving all of your crap.

      Comment


        #4
        http://www.szu.cz/svi/cejph/archiv/2011-3-09-
        full.pdf

        GEOGRAPHIC ACCUMULATION OF
        CREUTZFELDT- JAKOB DISEASE IN SLOVAKIA
        – ENVIRONMENTAL METAL IMBALANCE AS A
        POSSIBLE COFACTOR
        Dana Slivarichová1, Eva Mitrová1, Monika
        Ursínyová2, Iveta Uhnáková2, Silvia Koščová1,
        Ladislava Wsólová3
        1Department of Prion Diseases, Slovak Medical
        University, Bratislava, Slovak Republic
        2Laboratory of Toxic and Essential Elements,
        Slovak Medical University, Bratislava, Slovak
        Republic 3Department of Biostatistical Analysis,
        Slovak Medical University, Bratislava, Slovak
        Republic.

        More science looking at the actual diseased brain
        tissue - CJD patients/Slovakia. Free on-line.

        No problem with methodology of Vodyanyo, but
        after waiting for - 25 years... It would be nice to
        see which metals are prevalent in the BSE and
        CWD brain.

        Aberrant metal binding by prion protein in human
        prion disease
        Boon-Seng Wong,* Shu G. Chen,* Monica
        Colucci,*,1 Zhiliang Xie,* Tao Pan,* Tong Liu,*
        Ruliang Li,* Pierluigi Gambetti,* Man-Sun Sy* and
        David R. Brown2
        *National Prion Disease Pathology Surveillance
        Center, Institute of Pathology, Case Western
        Reserve University School of Medicine,
        Cleveland, Ohio, USA
        2Department of Biochemistry, University of
        Cambridge, Cambridge, UK

        http://ela-
        europe.org/ELA%20teksten/library/trace%20metal
        s/Aberrant%20metal%20binding%20by%20prion
        %20protein.pdf

        Also free on-line...

        Without the acknowledged research showing what
        metals are found in diseased brain tissue,
        researchers might look for years at metal effects
        with no correlation to a specific disease...

        Dr. Vodyanyoy acknowledges the past works of
        Brown, Wong, Bush and even Purdey; their
        results are guiding his research path for the
        future. I suggest reading these two papers for
        starters.

        Comment

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