I must ask our politicians and veterinary/CFIA officials, if they can read. I have provided them plenty of evidence from peer-reviewed medical papers as well as other documentation (including patents, university data, etc) on how the TSEs are a result of metal particles nucleating proteins.
Further to this, I wish to add a new "review" by D.Carleton Gajdusek. This man was the original researcher who looked at kuru in Papa New Guinea.
Link to paper: http://journals.royalsociety.org/content/2gu458u9763742w2/fulltext.pdf
This "review" or discussion paper, clearly points to what Gajdusek calls "amyloid-enhancing agents", such as AgN03, BEING the scrapie-like agent.
Please take time to read this very short document (only 4 pages).
When I get a chance, I'll write more about what he is discussing, in regards to nucleation, crystals, and metals.
Quotes from
"Kuru and its contributions to medicine" by D. Carleton Gajdusek.
"11. AMYLOID-ENHANCING FACTORS ARE
SCRAPIE-LIKE INFECTIONS/AMYLOID
NUCLEANTS
For approximately 35 years, I have been aware of the work of amyloidologists in their attempts to accelerate the appearance of AA amyloid deposits in animals primed with inoculation of AgNO3 or heterologous casein. Their discovery of 'amyloid-enhancing factors'
(Niewold et al. 1987), which were active in high dilutions and difficult to purify, reminded me of our
problems with the infectious agents of scrapie or kuru. I suggested that amyloid-enhancing factors were
scrapie-like agents (Gajdusek 1988, 1991, 1994a,b)."
amyloid-enhancing agents such as silver nitrate were [ARE] the agents causing disease.
"16. MONTMORILLONITE CLAY DEPOSITS LEAD
TO DELAYED NEURODEGENERATIVE DISEASES
The high-incidence foci of two very different diseases, Guamanian amyotrophic lateral sclerosis (lytico) and parkinsonism–dementia (bodig), occurred also in a few remote inland villages on Honshu Island in Japan and
among the Auyu and Jakai people around Bade and Kepi in southern West New Guinea (Gajdusek & Salazar 1982). It has virtually disappeared from all of these places with the introduction of civilization. These three foci were restricted to remote communities in which there was such a depletion of environmental calcium as to produce a chronic severe deficiency of calcium in
the diet, with the result that calcium sparing led to soft tissue deposition of calcium aluminium silicate or
montmorillonite clay deposits within brain cells, along with other heavy elements as the diet provided. These
lay dormant for decades until triggered later in life to cause specific neuronal damage leading to lytico or bodig. Civilization, with all its ills, has caused these two diseases in all the three locations to disappear."
Montmorillonite clay is a "calcium aluminum silicate" mineral. In the described regions, the areas with a low calcium level in soils, and an abundance of other "heavy elements", resulted in the development (over a long period of time) of neurological disease.
Prion researchers from Wisconsin who did experiments with montmorillonite clay, prions fragments and genetically altered mice, stated that the montmorillonite alone did not cause disease. However, it is now revealed that in a low calcium situation, calcium is replaced by a heavier element and it does result in disease (even without prion proteins). This leads me to ask, what heavier element replaced the calcium?
More evidence that the lack of a required metal, and its substitution with another heavier elements, can initiate disease processes.
Further to this, I wish to add a new "review" by D.Carleton Gajdusek. This man was the original researcher who looked at kuru in Papa New Guinea.
Link to paper: http://journals.royalsociety.org/content/2gu458u9763742w2/fulltext.pdf
This "review" or discussion paper, clearly points to what Gajdusek calls "amyloid-enhancing agents", such as AgN03, BEING the scrapie-like agent.
Please take time to read this very short document (only 4 pages).
When I get a chance, I'll write more about what he is discussing, in regards to nucleation, crystals, and metals.
Quotes from
"Kuru and its contributions to medicine" by D. Carleton Gajdusek.
"11. AMYLOID-ENHANCING FACTORS ARE
SCRAPIE-LIKE INFECTIONS/AMYLOID
NUCLEANTS
For approximately 35 years, I have been aware of the work of amyloidologists in their attempts to accelerate the appearance of AA amyloid deposits in animals primed with inoculation of AgNO3 or heterologous casein. Their discovery of 'amyloid-enhancing factors'
(Niewold et al. 1987), which were active in high dilutions and difficult to purify, reminded me of our
problems with the infectious agents of scrapie or kuru. I suggested that amyloid-enhancing factors were
scrapie-like agents (Gajdusek 1988, 1991, 1994a,b)."
amyloid-enhancing agents such as silver nitrate were [ARE] the agents causing disease.
"16. MONTMORILLONITE CLAY DEPOSITS LEAD
TO DELAYED NEURODEGENERATIVE DISEASES
The high-incidence foci of two very different diseases, Guamanian amyotrophic lateral sclerosis (lytico) and parkinsonism–dementia (bodig), occurred also in a few remote inland villages on Honshu Island in Japan and
among the Auyu and Jakai people around Bade and Kepi in southern West New Guinea (Gajdusek & Salazar 1982). It has virtually disappeared from all of these places with the introduction of civilization. These three foci were restricted to remote communities in which there was such a depletion of environmental calcium as to produce a chronic severe deficiency of calcium in
the diet, with the result that calcium sparing led to soft tissue deposition of calcium aluminium silicate or
montmorillonite clay deposits within brain cells, along with other heavy elements as the diet provided. These
lay dormant for decades until triggered later in life to cause specific neuronal damage leading to lytico or bodig. Civilization, with all its ills, has caused these two diseases in all the three locations to disappear."
Montmorillonite clay is a "calcium aluminum silicate" mineral. In the described regions, the areas with a low calcium level in soils, and an abundance of other "heavy elements", resulted in the development (over a long period of time) of neurological disease.
Prion researchers from Wisconsin who did experiments with montmorillonite clay, prions fragments and genetically altered mice, stated that the montmorillonite alone did not cause disease. However, it is now revealed that in a low calcium situation, calcium is replaced by a heavier element and it does result in disease (even without prion proteins). This leads me to ask, what heavier element replaced the calcium?
More evidence that the lack of a required metal, and its substitution with another heavier elements, can initiate disease processes.
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