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    #16
    Resistance is a complex topic and to make generalizations is dangerous. Get
    into disease resistance and it's even more complicated. We farmers can't
    even begin to understand it. It like talking genetics, after a very short
    time its just words - big ones!

    There are 2 main types of weed resistance, enhanced metabolism and target
    site.

    Enhanced metabolism. This causes more rapid herbicide detoxification
    within resistant plants. Resistance tends to be partial but plants may be
    cross resistant to a wide range of herbicides with different modes of
    action. This form of resistance tends to develop quite slowly over a period
    of years. Roundup falls in this category.

    Target site resistance. This blocks the site of activity specific to the
    herbicide’s mode of action. This usually results in complete resistance to
    herbicides acting on that specific site, but not to herbicides acting on
    different targets. Two main types have been identified: one affects ‘fop’
    and ‘dim’ graminicides (ACCase inhibitors) - Group 1; the other affects
    sulfonylurea herbicides (ALS inhibitors) - Group 2. Resistant populations
    can increase rapidy.

    This post is about Group 1 target site resistance and is what I was
    referring to.

    A good read:

    http://www.umanitoba.ca/outreach/naturalagriculture/weed/files/herbicide/re
    duced_rates_e.htm


    If a wild oat biotype is resistant, it can easily take 40 times the rate to
    kill it as there is no target site in the plant for the ACCase herbicide to
    do its job. That's when you hope the liberty is getting it.

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