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Tam says the federal government is 'examining' all vaccine mandates

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    #16
    Okay what ever you say there A5.

    You should write the journal that she publishes her study in and tell her she's wrong that covid is good and don't worry about long covid and neurological damage. Say it's all good based on your home science kit and some good old Alberta ingenuity, speculation and home research!

    I am sure they will write back with praise for your astute observations. You may even be awarded the Nobel prize in neuroscience.

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      #17
      Originally posted by chuckChuck View Post
      Okay what ever you say there A5.

      You should write the journal that she publishes her study in and tell her she's wrong that covid is good and don't worry about long covid and neurological damage. Say it's all good based on your home science kit and some good old Alberta ingenuity, speculation and home research!

      I am sure they will write back with praise for your astute observations. You may even be awarded the Nobel prize in neuroscience.
      It's almost as an if we are having a completely different conversation.
      Either that or else you can't be bothered reading what I post.

      Comment


        #18
        Here A5 now you can take a look at the science in "question" authored by numerous researchers working in neurology not just one who speculated that covid which clearly caused severe damage to organs, also could cause brain damage. She then worked with her colleagues to prove or disprove her hypothesis using the scientific method. Was she and her numerous colleagues wrong? If you think so, show us the evidence! LOL

        https://www.biorxiv.org/content/10.1101/2022.01.07.475453v1

        Mild respiratory SARS-CoV-2 infection can cause multi-lineage cellular dysregulation and myelin loss in the brain

        Anthony Fernández-Castañeda, Peiwen Lu, Anna C. Geraghty, Eric Song, Myoung-Hwa Lee, Jamie Wood, Belgin Yalçın, Kathryn R. Taylor, Selena Dutton, Lehi Acosta-Alvarez, Lijun Ni, Daniel Contreras-Esquivel, Jeff R. Gehlhausen, Jon Klein, Carolina Lucas, Tianyang Mao, View ORCID ProfileJulio Silva, Mario A. Peña-Hernández, Alexandra Tabachnikova, Takehiro Takahashi, Laura Tabacof, Jenna Tosto-Mancuso, Erica Breyman, Amy Kontorovich, Dayna McCarthy, Martha Quezado, Marco Hefti, Daniel Perl, Rebecca Folkerth, David Putrino, View ORCID ProfileAvi Nath, View ORCID ProfileAkiko Iwasaki, View ORCID ProfileMichelle Monje

        Abstract

        Survivors of Severe Acute Respiratory Syndrome Coronavirus-2 (SARS-CoV-2) infection frequently experience lingering neurological symptoms, including impairment in attention, concentration, speed of information processing and memory. This long-COVID cognitive syndrome shares many features with the syndrome of cancer therapy-related cognitive impairment (CRCI). Neuroinflammation, particularly microglial reactivity and consequent dysregulation of hippocampal neurogenesis and oligodendrocyte lineage cells, is central to CRCI. We hypothesized that similar cellular mechanisms may contribute to the persistent neurological symptoms associated with even mild SARS-CoV-2 respiratory infection. Here, we explored neuroinflammation caused by mild respiratory SARS-CoV-2 infection – without neuroinvasion - and effects on hippocampal neurogenesis and the oligodendroglial lineage. Using a mouse model of mild respiratory SARS-CoV-2 infection induced by intranasal SARS-CoV-2 delivery, we found white matter-selective microglial reactivity, a pattern observed in CRCI. Human brain tissue from 9 individuals with COVID-19 or SARS-CoV-2 infection exhibits the same pattern of prominent white matter-selective microglial reactivity. In mice, pro-inflammatory CSF cytokines/chemokines were elevated for at least 7-weeks post-infection; among the chemokines demonstrating persistent elevation is CCL11, which is associated with impairments in neurogenesis and cognitive function. Humans experiencing long-COVID with cognitive symptoms (48 subjects) similarly demonstrate elevated CCL11 levels compared to those with long-COVID who lack cognitive symptoms (15 subjects). Impaired hippocampal neurogenesis, decreased oligodendrocytes and myelin loss in subcortical white matter were evident at 1 week, and persisted until at least 7 weeks, following mild respiratory SARS-CoV-2 infection in mice. Taken together, the findings presented here illustrate striking similarities between neuropathophysiology after cancer therapy and after SARS-CoV-2 infection, and elucidate cellular deficits that may contribute to lasting neurological symptoms following even mild SARS-CoV-2 infection.
        Last edited by chuckChuck; Mar 24, 2022, 07:48.

        Comment


          #19
          Once again Chuck, you are having a different discussion. I'm not arguing the outcomes or denying the long term side effects of Covid. Stop putting words in my mouth, or the doctors mouth.
          I'm pointing out that the Doctor specifically stated what the outcomes would be before there was the possibility of any evidence to support any such conclusion. That doesn't mean she was wrong, or that there would be no long term side effects.
          I'm just pointing out that this isn't how ethical science works. You don't start out with a predetermined outcome then design a study to prove it, unless of course it is in the field of climate science, where the rot seems to have spilled over into other previously ethical fields of science.

          Try reading and comprehending what other people post before going off on an incoherent rant about unrelated topics.

          Comment


            #20
            "You don't start out with a predetermined outcome then design a study to prove it," Huh?

            Hypothesis. A supposition or proposed explanation made on the basis of limited evidence as a starting point for further investigation.

            If you are going to make this allegation then you should have some evidence other than her informal hypothesis in the media that she and her colleagues designed a study to come to a predetermined outcome.

            Because that's not likely the case.

            Comment


              #21
              Originally posted by chuckChuck View Post
              "You don't start out with a predetermined outcome then design a study to prove it," Huh?

              Hypothesis. A supposition or proposed explanation made on the basis of limited evidence as a starting point for further investigation.

              If you are going to make this allegation then you should have some evidence other than her informal hypothesis in the media that she and her colleagues designed a study to come to a predetermined outcome.

              Because that's not likely the case.
              It is only a direct quote from the author.

              Comment


                #22
                So no evidence that the study was designed to deliver the desired outcome? Figured as much! LOL

                You should really pick an argument you can win sometime with some actual evidence.
                Last edited by chuckChuck; Mar 24, 2022, 08:23.

                Comment

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